Publikationen

2010

• C/EBPbetaDeltauORF mice--a genetic model for uORF-mediated translational control in mammals.
  Wethmar K, Bégay V, Smink JJ, Zaragoza K, Wiesenthal V, Dörken B, Calkhoven CF, Leutz A
  Genes Dev 2010, 24(1), 15-20
  
• A poke into the diversity and associations within human anterior nare microbial communities.
  Wos-Oxley ML, Plumeier I, von Eiff C, Taudien S, Platzer M, Vilchez-Vargas R, Becker K, Pieper DH
  ISME J 2010, 4(7), 839-51
  
• Comparative gene expression in toxic versus non-toxic strains of the marine dinoflagellate Alexandrium minutum.
  Yang I, John U, Beszteri S, Glöckner G, Krock B, Goesmann A, Cembella AD
  BMC Genomics 2010, 11, 248
  
• Molecular characterization of nuclear DNA helicase II (RNA helicase A).
  Zhang S, Grosse F
  Methods Mol Biol 2010, 587, 291-302
  
• Genomic analysis of the 55 kDa subunit of DNA polymerase epsilon in human intracranial neoplasms.
  Zhou Q, Lehtonen T, Lehtonen J, Laakso S, Szymas J, Pospiech H, Huoponen K, Syväoja J, Collan Y
  Cancer Genomics Proteomics 2010, 7(3), 143-6
  
• Characterization of mesonephric development and regeneration using transgenic zebrafish.
  Zhou W, Boucher RC, Bollig F, Englert C, Hildebrandt F
  AM J PHYSIOL-RENAL 2010, 299(5), F1040-7
  

2009

• Bacterial L-forms.
  Allan EJ, Hoischen C, Gumpert J
  In: Advances in Applied Microbiology 2009, 68, 1-39, Elsevier Inc., New York, N
  
• Exogenous brain-derived neurotrophic factor (BDNF) reverts phenotypic changes in the retinas of transgenic mice lacking the BDNF gene.
  Arango-González* B, Cellerino* A, Kohler K
  Invest Ophthalmol Vis Sci 2009, 50(3), 1416-22 * equal contribution, published during change of institution
  
• Conditional inactivation of the NBS1 gene in the mouse central nervous system leads to neurodegeneration and disorganization of the visual system.
  Baranes K, Raz-Prag D, Nitzan A, Galron R, Ashery-Padan R, Rotenstreich Y, Assaf Y, Shiloh Y, Wang ZQ, Barzilai A, Solomon AS
  Exp Neurol 2009, 218(1), 24-32
  
• The prolyl cis/trans isomerase cyclophilin 18 interacts with the tumor suppressor p53 and modifies its functions in cell cycle regulation and apoptosis.
  Baum N, Schiene-Fischer C, Frost M, Schumann M, Sabapathy K, Ohlenschläger O, Grosse F, Schlott B
  Oncogene 2009, 28(44), 3915-25